Biology of oral cancer [electronic resource] : key apoptotic regulators / Prakash S. Bisen, Zakir Khan, Saurabh Bundela.

By:

Bisen, Prakash S

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Material type: Text

TextPublication details: Boca Raton : CRC Press, 2014.Description: xix, 272 p. : illISBN:

9781466575622 (ebook : PDF)

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Electronic books.

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Also available in print edition.

Contents:

ch. 1. Cancer : a worldwide menace -- ch. 2. Oral cancer -- ch. 3. Proliferative and apoptotic signaling in oral cancer -- ch. 4. Apoptotic regulations -- ch. 5. Dynamics of p53 in oral cancer -- ch. 6. Diagnostic and therapeutic potential of apoptotic marker -- ch. 7. Expression and regulation of Survivin -- ch. 8. Therapeutics of Survivin -- ch. 9. Molecular diagnosis of oral cancer.

Summary: "Cancer is a class of disease or disorders characterized by uncontrolled division of cells and the ability of these cells to invade other tissues. One new approach in dealing with cancer is through apoptotic regulators, which is the process of controlling cell growth by abnormally prolonging cell survival, facilitating the accumulation of transforming mutations and promoting resistance. This book uses oral cancers as the specific means to demonstrate this technique with this particular type of cancer, which is quite prevalent (and deadly) in India and other parts of Asia, in particular"--Provided by publisher.

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Includes bibliographical references and index.

"Cancer is a class of disease or disorders characterized by uncontrolled division of cells and the ability of these cells to invade other tissues. One new approach in dealing with cancer is through apoptotic regulators, which is the process of controlling cell growth by abnormally prolonging cell survival, facilitating the accumulation of transforming mutations and promoting resistance. This book uses oral cancers as the specific means to demonstrate this technique with this particular type of cancer, which is quite prevalent (and deadly) in India and other parts of Asia, in particular"--Provided by publisher.

Also available in print edition.

Mode of access: World Wide Web.

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